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naproxen (naproxen, Verex / naproxen, Biovail)

✓ Approved

Bausch Health Companies Inc. · PTGS1 · Small Molecule

What is naproxen?

naproxen is a small molecule developed by Bausch Health Companies Inc.. It is approved for therapeutic indications via oral (po).

Drug Profile

Brand Namesnaproxen, Verex, naproxen, Biovail
CompanyBausch Health Companies Inc.
Drug ClassSmall Molecule
Molecular TargetPTGS1, PTGS2
RouteOral (PO)
StatusApproved

Mechanism of Action

Molecular Targets

naproxen acts on 2 molecular targets:

PTGS1prostaglandin-endoperoxide synthase 1 (COX3, PCOX1)
PTGS2prostaglandin-endoperoxide synthase 2 (GRIPGHS, hCox-2)
Want deeper analysis?Noah AI can explain complex mechanisms and compare to similar drugs.

Therapeutic Indications

naproxen is developed for 1 unique indication across 1 therapeutic area.

Therapeutic AreaConditionPhase
Musculoskeletal and connective tissue disordersMusculoskeletal pain✓ Approved

Related Research Articles

PubMedChemMedChem2026-07-16

Computer-Aided Drug Design, Synthesis, and In Vitro Safety Evaluation of Carprofen Analogs for Novel Alzheimer's Disease Therapeutics.

Acosta-Guzmán Paola P, Bedoya-Malagón Daniel D, Mercado-Coy Luisa L, Morantes Sandra Johanna SJ et al.

Nonsteroidal anti-inflammatory drugs (NSAIDs), such as ibuprofen, naproxen, and indomethacin, have reduced amyloid beta (Aβ) levels and improved cognitive function in mouse models of Alzheimer's disease (AD). The objective of the current research is to evaluate different NSAIDs using computer-aided drug design (CADD) to explore their potential as new treatments for AD. CADD is an advanced technique for designing biologically active molecules and involves two primary approaches: structure-based virtual screening and ligand-based virtual screening. After identifying the NSAID with the highest affinity for amyloid Aβ1-42 fibrils (PDB 2BEG), we designed analogs through bioisosteric modifications. Twenty derivatives were created, and two carprofen analogs with the most promising properties were synthesized, yielding 75% success with compound A-1 and 70% with compound A-2. These analogs exhibited lower predicted IC50 values (1.57 μM) and more negative interaction energies (-6.43 and -6.67 kcal/mol for A-1 and A-2, respectively) compared to carprofen. Safety evaluations in L929 cells indicated that these compounds were safe at concentrations of 40 µM. In conclusion, two carprofen analogs were successfully designed and synthesized, demonstrating higher affinity for the amyloid Aβ1-42 fibrils, better-predicted IC50 values than carprofen, and safety in the L929 in vitro assay.

PubMedZygote (Cambridge, England)2026-07-15

The interruption of the treatment with naproxen restored the C57BL/6 ovulation rate.

Castro João Pedro Lobão Gomes JPLG, Guimarães Vanessa Neves VN, Barbosa Patrick de Abreu PA, Paz Amanda Pereira AP et al.

Naproxen is a widely used nonsteroidal anti-inflammatory drug, and previous studies have shown that its administration in mice influenced antral follicle growth and ovulation. Therefore, the present study aimed to investigate the reversibility of these effects following treatment interruption and to evaluate their impact across different mouse backgrounds. For Experiment I, C57BL/6 females (n = 20, 6 weeks of age) were treated with 10 mg/kg (low, n = 7) or 50 mg/kg (high, n = 7) of naproxen, while animals of the vehicle group received PBS + DMSO 5% (n = 6), followed by 21 days of rest. After euthanasia, the ovaries were histologically analysed. For Experiment II (C57BL/6) and Experiment III (Swiss and BALB/c), animals were treated for eight days with the same protocol, receiving 20 IU of eCG and 20 IU of hCG for ovulation induction. Recovered oocytes were collected and quantified. Results demonstrated that the treatment affected the percentage of primary follicles, which increased in the high-dose group compared with the vehicle group. Additionally, the rate of antral follicles was higher in the high group than in the vehicle and low-dose groups. The number of ovulated oocytes was not altered in BALB/c and Swiss females, as well as the number of corpora haemorrhagica and corpus luteum (p > 0.05). Overall, our findings indicate that naproxen treatment may have transient effects on folliculogenesis in C57BL/6 mice, with no impact in BALB/c or Swiss mice.

PubMedBioprocess and biosystems engineering2026-07-13

A Pichia pastoris-based platform for large-scale production of functional recombinant human serum albumin with therapeutic efficacy.

Li Lijiong L, Lin Jing J, Cui Mengfa M, Fu Hailong H et al.

Human serum albumin (HSA) is critical for treating liver cirrhosis with ascites. However, plasma-derived HSA (pHSA) faces limitations in supply and carries potential infection risks. To address the urgent need for a therapeutically equivalent alternative, we established an efficient expression system for recombinant HSA (rHSA) in Pichia pastoris. We purified rHSA via a three-step chromatography purification process, yielding approximately 260 mg per 1.5 L of culture. Comprehensive analyses confirmed its structural equivalence to pHSA. Mass spectrometry revealed a molecular weight of 66,530.8 Da, while circular dichroism showed a 36.9% α-helical content. Furthermore, isothermal titration calorimetry demonstrated comparable drug-binding affinities for naproxen and warfarin. In a CCl₄-induced rat model of liver cirrhosis with ascites, rHSA displayed significant, dose-dependent therapeutic effects. Notably, high-dose rHSA achieved efficacy statistically equivalent to pHSA. It reduced ascites volume by 50.1%, improved liver index by 33.5% and restored colloid osmotic pressure along with liver function markers (ALT, AST, and bilirubin). This study provides the first in vivo evidence confirming the therapeutic equivalence and dose-effect relationship of P. pastoris-derived rHSA compared with pHSA. These findings establish a scalable, cost-effective, and safe platform for rHSA production, supporting its potential as a viable clinical substitute for plasma-derived albumin.

PubMedmedRxiv : the preprint server for health sciences2026-07-10

NSAID use is associated with lower dementia and Alzheimer's disease prevalence and slower cognitive decline: A retrospective longitudinal analysis of the NACC cohort.

Hoehne Carolin Luisa CL, Salinas Victor V, Shirani Afsaneh A, Stuve Olaf O et al.

Dementia, particularly Alzheimer's disease (AD), is a major global health challenge, with prevalence projected to reach 150 million cases by 2050. AD is characterized by progressive cognitive decline linked to neuroinflammation and neurodegeneration. Non-steroidal anti-inflammatory drugs (NSAIDs) have been explored as potential neuroprotective agents, particularly diclofenac, which has been proposed to modulate microglial inflammasome signaling. However, prior studies investigating NSAIDs in AD have yielded inconsistent findings. We therefore reexamined the relationship between selected NSAIDs and dementia outcomes in a large longitudinal cohort from the National Alzheimer's Coordinating Center (NACC). We analyzed cross-sectional and longitudinal data from the NACC database collected between 2005 and 2022. Associations between NSAID exposure and dementia, AD, and cognitive trajectories were examined. Propensity score matching was performed to compare NSAID users with matched non-users while adjusting for demographic and clinical confounders. Longitudinal mixed-effects models were used to assess cognitive decline based on Montreal Cognitive Assessment (MoCA) scores. Among 47,165 participants, diclofenac and naproxen use were associated with a lower prevalence of dementia and AD compared with matched non-users, whereas etodolac showed no significant associations. Diclofenac users demonstrated reduced odds of dementia and AD. Naproxen showed similar cross-sectional associations. In longitudinal modeling, diclofenac users had a significantly slower rate of cognitive decline than non-users. These findings suggest a compound-specific association between NSAID use and AD, with diclofenac potentially modulating disease progression through anti-inflammatory mechanisms. The observed modulation of longitudinal cognitive decline supports further investigation of inflammatory pathways, including microglial and inflammasome signaling, as therapeutic targets in biomarker-defined AD populations.

PubMedWater research2026-07-09

Metagenomic insights into microbial drivers of organic micropollutant removal in wastewater-impacted riverbank filtration.

Zhai Yujia Y, Wang Xun X, Deng Xuhan X, Li Xiaoming X et al.

Organic micropollutants (OMPs) in wastewater treatment plant (WWTP) effluent pose persistent risks to aquatic ecosystems and drinking water sources. Riverbank filtration (RBF) is a nature-based treatment process, yet the compartment-specific roles of riverbed sediment and downstream soil in OMP attenuation remain poorly resolved under wastewater-impacted conditions. Here, we combined targeted chemical analysis, OMP property compilation, shotgun metagenomics, EnviPath-based biotransformation annotation, and exploratory network analysis to investigate OMP attenuation in a laboratory-scale RBF system treating real WWTP effluent for 10 months. Nineteen OMPs were monitored along a sequential sediment-soil filtration pathway. Sediment preferentially attenuated hydrophilic or charged compounds, including lidocaine, amantadine, and sotalol, whereas soil contributed more strongly to the attenuation of naproxen, atenolol, and losartan. Metagenomic profiling revealed distinct microbial communities and functional gene repertoires between sediment and soil after long-term operation. Sediment harbored higher relative abundances of genes associated with oxidative xenobiotic transformation, including cytochrome P450-related enzymes, demethylases, dehydrogenases, oxidases, and aromatic compound degradation pathways. An exploratory Spearman network further identified associations among microbial genera, EnviPath-annotated candidate biotransformation genes, and OMP removal rates, including 17 KO-OMP links supported by both correlation and pathway annotation. These findings indicate that sediment and soil develop complementary microbial functional potentials that may support compound-specific OMP attenuation. This study provides a mechanistic basis for optimizing sediment-soil configurations in wastewater-impacted RBF systems and for improving nature-based barriers against diverse OMP mixtures.

PubMedInorganic chemistry2026-07-08

Tuning Au Reactivity Beyond Canonical Targets: Ligand-Driven Au(I) Metalation of Lysine Residues in Hen Egg White Lysozyme.

Piroddu Davide D, Famlonga Luca L, Tolbatov Iogann I, Ferraro Giarita G et al.

Au(I) complexes are widely investigated as therapeutic agents due to their high affinity for biological nucleophiles and protein targets. Here, the reactivity of an Auranofin (AF) derivative bearing naproxen as a ligand toward hen egg white lysozyme (HEWL) was investigated by a combined crystallographic and computational approach. Notably, the first observation of lysine metalation in HEWL by an Au complex is reported. The results highlight how ligand substitution can significantly affect Au(I) reactivity toward biomacromolecules, enabling noncanonical targeting and potentially impacting biological activity.

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