Drug Database
YY

YY-3110 (YY 3110 / YY3110)

✓ Approved

YuYu Pharma · Small Molecule · Small Molecule

What is YY-3110?

YY-3110 is a small molecule developed by YuYu Pharma. It is approved for therapeutic indications via unknown.

Drug Profile

Brand NamesYY 3110, YY3110
CompanyYuYu Pharma
Drug ClassSmall Molecule
RouteUnknown
StatusApproved

Therapeutic Indications

YY-3110 is developed for 1 unique indication across 1 therapeutic area.

Therapeutic AreaConditionPhase
Vascular disordersThrombosis✓ Approved

Related Research Articles

PubMedGenome medicine2026-07-17

A short-term randomized clinical trial testing feasibility of and physiological responses to a Ruminococcus torques strain in healthy overweight humans.

Gæde Joachim J, Fan Yong Y, Lyu Liwei L, Stankevic Evelina E et al.

The human gut microbiota interacts with host biology as exemplified by the effects of the Ruminococcus torques (RT) ATCC 27756 strain-a commensal human intestinal bacterial strain that synthesizes the polypeptides RORDEP1 and RORDEP2 which in rodents improve metabolism. Motivated by the metabolic effects of the RT strain in the rodent host, we explored in a short-term double blind and placebo-controlled randomized cross-over trial the feasibility and potential physiological responses of the same bacterial strain in humans. The trial undertaken at Herlev and Gentofte Hospital, Denmark included 32 healthy overweight adults. Decided by block randomization with blocks of six, we infused either 3.1 × 1011 colony forming units of the live RT ATCC 27756 strain or placebo into the duodenum during an observation period of six hours including a two-hour oral glucose tolerance test. Insulin sensitivity measured as Matsuda Insulin Sensitivity Index was the primary endpoint. The infusion of the bacterium was safe and well-tolerated. We found no effects on the primary endpoint of the trial. Compared to placebo, short-term RT infusion induced a relative rise in plasma concentrations of glucagon-like-peptide-1 (GLP-1) and peptide YY (PYY) in parallel with a relative decline in gastric inhibitory polypeptide (GIP). These intestinal hormone responses mirrored those previously reported from studies in rats. The abundance of secondary bile acids in plasma as well as a plasma marker of bone remodeling increased after infusion of RT compared to placebo. Measures of glucose tolerance, energy expenditure, cutaneous thermography, and main markers of systemic low-grade inflammation remained unchanged. Duodenal infusion during six hours of the RT ATCC 27756 strain in healthy overweight humans shows that the bacterial strain is well tolerated, and the short-term effects on intestinal hormone release align with those previously reported in rodents. The trial is registered prospectively in ClinicalTrials.gov on 2022-06-28 with ID NCT05448274 (https://clinicaltrials.gov/study/NCT05448274?intr=ruminococcus%20torques&viewType=Card&rank=1).

PubMedNature reviews. Endocrinology2026-07-15

Lipid sensing and brain hormone receptors in food intake and glucose regulation.

Garrido Ameth N AN, Lyons Sulayman Aslan SA, Moslemian Dorsa D, Beaudry Jacqueline L JL et al.

The body possesses critical nutrient sensors that rapidly coordinate feeding behaviour and systemic metabolism through hormones and neural pathways. In this Review, we highlight the mechanisms by which lipid sensing triggers the release of small intestinal-derived cholecystokinin, peptide YY, glucagon-like peptide 1 and glucose-dependent insulinotropic polypeptide, as well as kidney-derived growth differentiation factor 15, to regulate satiety and glucose homeostasis through the brain. We discuss how dysregulation of lipid sensing pathways contributes to obesity and type 2 diabetes mellitus, and how interventions including bariatric surgery, modulation of the gut microbiota and pharmacological agonists restore polyhormonal secretion and action on metabolism. We propose that lipid sensing in the small intestine and the kidney orchestrates an endocrine and neural axis that governs energy balance, providing a lipid-sensing-dependent framework for the development of next-generation therapies in obesity and metabolic disease to remotely and concurrently target multiple brain hormone receptors to lower food intake and body weight.

PubMedEuropean journal of clinical nutrition2026-07-15

Acute appetite and gut-hormone responses to two combined training sessions: a randomized crossover study in men with obesity.

Li Nan N, Wu Min M, Li Yanchun Y, Yang Xiangang X

This randomized crossover study compared acute appetite-related, gut-hormone, and energy-intake responses between moderate-intensity resistance plus aerobic training (RAT) and high-intensity functional plus aerobic training (FAT) in males with obesity. Twenty-one men with obesity completed both protocols in a randomized crossover design. Subjective appetite was assessed using a visual analogue scale, and insulin, glucagon-like peptide-1, and peptide YY (PYY) were measured pre-, post-, and 1 h post-exercise. Energy intake was recorded during subsequent meals and over 2 days. PYY responses differed between conditions over time, with a significant condition × time interaction (P = 0.015). At 1 h post-exercise, PYY concentrations were higher after RAT than after FAT (ratio = 1.69, 95% CI 1.26-2.27; adjusted P < 0.001). PYY AUC was higher in RAT than in FAT (ratio [RAT/FAT] = 1.44; P = 0.016). Fullness AUC was lower after RAT than after FAT, whereas prospective food consumption AUC was higher after RAT than after FAT (Δ [RAT - FAT] = -670.0 and 701.4 mm·120 min; P = 0.014 and P = 0.016, respectively). No between-condition differences were observed in energy intake on the trial day or the following day. Varying the resistance component within combined exercise may elicit distinct acute hormonal and perceptual appetite responses in young men with obesity, without altering short-term energy intake. These findings should be interpreted as comparative responses between RAT and FAT rather than exercise-induced effects per se.

PubMedMolecular therapy : the journal of the American Society of Gene Therapy2026-07-09

Distinct YY dinucleotide periodicity in Adeno-associated virus (AAV) DNA.

Baumgartl Conradin C, Becker Jonas J, Naber Lena L, Xu Man M et al.

Dinucleotide periodicity is a hallmark of genome organization, yet its role in single-stranded (ss)DNA viruses remains poorly understood. Here, we systematically analyzed dinucleotide spacing patterns in Adeno-associated virus (AAV) genomes and other viruses. Across 13 primate AAV serotypes, we identify a pronounced and highly conserved ∼15 bp periodicity specific to pyrimidine-pyrimidine (YY) dinucleotides and their reverse complements (RR). Comparative analyses across >25000 viral sequences demonstrate that this 15-bp YY/RR periodicity is unique to the genus Dependoparvovirus and absent from other ssDNA viruses, satellite viruses, and helper viruses, which predominantly exhibit canonical ∼10-11 bp periodicities. Upon disruption of the YY/RR pattern using DNA family shuffling of AAV capsid genes, and subsequent iterative selection for viral production or cell entry, we found that the pattern is under positive selection. Selected sequences display increased periodicity alongside reduced sequence diversity, supporting a functional role for this genomic feature. Finally, engineered recombinant AAV genomes containing YY-periodic motifs exhibit enhanced production and, for some designs, improved transduction efficiency, demonstrating that YY periodicity can modulate viral replication and infectivity. Our findings uncover a unique DNA-encoded signal in dependoparvoviruses that contributes to AAV fitness, expands our knowledge of virus biology and has implications for vector engineering.

PubMedJournal of microbiology and biotechnology2026-07-09

Comparative Effects of Live and Heat-Killed Lacticaseibacillus paracasei HP7 on Intestinal Motility, Barrier-Related Markers, and Gut Microbiota in Delayed Transit Mice.

Gwon Hyeonjun H, Kim Hyeonji H, Kim Joo-Yun JY, Shim Jae-Jung JJ et al.

This study evaluated the effects of live and heat-killed Lacticaseibacillus paracasei HP7 on intestinal motility, host physiological responses, and gut microbiota in a mouse model of delayed intestinal transit. BALB/c mice were treated with live HP7 (HP7L), heat-killed HP7 (HP7K), or bisacodyl following loperamide administration. HP7L and HP7K improved intestinal motility-related parameters, including fecal output, stool consistency, fecal moisture content, and charcoal meal transit. HP7 treatment also modulated circulating gastrointestinal hormones and inflammatory cytokines, with changes in serotonin, ghrelin, peptide YY, IL-6, and IL-17A. In intestinal tissues, HP7 regulated the expression of genes related to neuroendocrine signaling, water transport, mucus production, and tight junction integrity, including Ghrl, Tac1, Slc6a4, Aqp3, Muc2, Tjp1, Tjp2, and Ocln. Histological analysis further showed reduced colonic tissue alterations and improved structural parameters. Microbiota analysis revealed that HP7L exerted broader effects on microbial community structure, whereas HP7K induced more selective taxonomic changes. Predicted functional profiling suggested that both HP7L and HP7K were associated with shifts in carbohydrate metabolism-related pathways, with HP7K showing additional predicted enrichment of pathways related to redox-associated metabolism, lipid metabolism, and microbial substrate utilization. These findings suggest that heat-killed HP7 retains biological activity and has potential as a postbiotic candidate for regulating intestinal motility.

PubMedmedRxiv : the preprint server for health sciences2026-07-03

Genetic and maternal environmental contributions to estimated fetal weight at 20 weeks gestation compared with birthweight.

Purdy Rosie A RA, Feng Jingxue J, Luo Zhenyu Z, Beaumont Robin N RN et al.

Birthweight reflects fetal growth from conception. The complications arising from the extremes of fetal growth are well known, but addressing these effectively depends on understanding how and when fetal growth is affected by various maternal and fetal factors. We aimed to compare associations of fetal genetic and maternal environmental factors with fetal weight mid-pregnancy and birthweight. We studied 3110 mother-child pairs from three population-based birth cohorts (UK and China). Estimated fetal weight at ~20 weeks gestation (EFW20) and birthweight were the outcomes of interest. Exposures were fetal genetic factors (fetal sex and fetal birthweight genetic score [BW GS]) and maternal factors (maternal BW GS, BMI, fasting plasma glucose [FPG], smoking, age, and parity). Associations were studied using multivariable linear regressions within cohorts and meta-analysed. All exposures were associated with both EFW20 and birthweight, apart from maternal FPG and smoking which were associated with birthweight only. Male fetal sex and a higher maternal BW GS were consistently associated with higher EFW20 and birthweight, whereas the fetal BW GS, maternal FPG, BMI and smoking showed more marked associations later in pregnancy. Maternal age and parity showed directionally opposite associations with EFW20 and birthweight. Fetal genetic and maternal environmental factors vary in their effect on fetal growth in mid-pregnancy compared with late pregnancy. These findings contribute to our understanding of growth across pregnancy and may inform the timing of clinical monitoring of fetal growth and interventions targeting modifiable maternal factors.

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