Drug Database
AL

alendronate (Binosto / Steovess / EX101)

✓ Approved

Ahn-Gook Pharmaceutical · Small Molecule · Small Molecule

What is alendronate?

alendronate is a small molecule developed by Ahn-Gook Pharmaceutical. It is approved for therapeutic indications via oral (po).

Drug Profile

Brand NamesBinosto, Steovess, EX101
CompanyAhn-Gook Pharmaceutical
Drug ClassSmall Molecule
RouteOral (PO)
StatusApproved

Therapeutic Indications

alendronate is developed for 3 unique indications across 2 therapeutic areas.

Therapeutic AreaConditionPhase
Musculoskeletal and connective tissue disordersOsteoporosis✓ Approved
Congenital, familial and genetic disordersHypertrophic cardiomyopathyPhase III
Musculoskeletal and connective tissue disordersOsteoarthritisPhase II

Related Research Articles

PubMedMediterranean journal of rheumatology2026-09-18

Cardiovascular Safety Signals of Osteoporosis Therapies: A FAERS Pharmacovigilance Study.

Sondhi Manush M, Singh Namrata N, Carkin Julie J, Hughes Grant G

To evaluate the associations between commonly used osteoporosis medications and major adverse cardiovascular events (MACE) using data from the Food and Drug Administration (FDA) Adverse Event Reporting System (FAERS). We conducted a retrospective pharmacovigilance analysis of FAERS reports between January 2019 and December 2024, identifying all reports involving alendronate, risedronate, zoledronic acid, denosumab, teriparatide, abaloparatide, or romosozumab. MACE outcomes included myocardial infarction (MI), stroke, atrial fibrillation (A-fib), and cardiac failure (CF), defined using Medical Dictionary for Regulatory Activities Preferred Terms. Disproportionality was assessed using reporting odds ratios (RORs), with signals defined by a lower 95% confidence interval >1. All analyses were descriptive and hypothesis-generating. FAERS contained more than 30 million adverse event reports during the study period. Romosozumab demonstrated the highest ROR for MI (2.38) and stroke (3.72), and also showed elevated signals for A-fib and CF (ROR 3.76). Zoledronic acid showed notable associations with stroke (ROR 2.42) and A-fib (ROR 3.22). Denosumab and abaloparatide were associated with the lowest RORs across all cardiovascular outcomes. Alendronate and risedronate demonstrated intermediate disproportionality signals, particularly for A-fib and CF. Romosozumab and zoledronic acid demonstrated prominent cardiovascular disproportionality signals in FAERS, whereas denosumab, abaloparatide, and teriparatide showed more favorable reporting profiles. These findings should not discourage osteoporosis treatment but underscore the importance of individualised cardiovascular risk assessment and the need for further prospective evaluation to inform safe therapy selection.

PubMedJCEM case reports2026-09-17

Medication-related osteonecrosis of the external auditory canal and treatment with teriparatide.

Xia Daheng D, Ting Matthew J M MJM, Atlas Marcus D MD, Boeddinghaus Rudolf R et al.

Medication-related osteonecrosis of the external auditory canal (MROEAC) is a rare complication of antiresorptive therapy, and guidance for diagnosis and management remains limited. We describe 2 patients with prolonged antiresorptive exposure who developed MROEAC and were treated with teriparatide. An 81-year-old woman developed bilateral external auditory canal erosions after 10 years of denosumab therapy. An 8-week course of teriparatide was associated with clinical improvement and a transient rise in bone formation markers, but computed tomography findings remained unchanged. Denosumab withdrawal led to delayed rebound bone resorption, prompting denosumab reintroduction. A 67-year-old woman developed left-sided MROEAC after 15 years of alendronate followed by a short course of denosumab. Four months of teriparatide was associated with symptomatic and biochemical improvement, but radiologic abnormalities persisted, and definitive surgery was required. These cases highlight the difficulty of differentiating MROEAC from more common otologic disorders, the discordance between clinical, biochemical, and radiologic response, the importance of multidisciplinary management, and therapeutic challenges created by denosumab discontinuation. Teriparatide was well tolerated and was associated with increased bone turnover markers, but radiologic resolution was incomplete. Surgical management may still be necessary.

PubMedBone2026-09-16

Alendronate alters mineral composition and has potential to restore enthesis strength without preventing trabecular bone loss during unloading of the supraspinatus enthesis.

Whittaker Sydney S, De Bruyker Isabelle I, Hemanth Neha N, Deymier Alix C AC

Prolonged unloading of the shoulder, most seen after stroke related hemiparesis, accelerates degeneration of the supraspinatus enthesis in part through localized bone loss at the tendon to bone interface. Because bisphosphonates are often used in populations experiencing disuse, it remains unclear whether they can mitigate bone deterioration at the supraspinatus enthesis. In this study, we examined how unloading and alendronate treatment influence bone architecture, mineral composition, cellular activity, and mechanical behavior at the supraspinatus enthesis using a 24-day murine model of botulinum toxin induced supraspinatus paralysis. Unloading produced significant trabecular deterioration, including reduced bone volume fraction and trabecular thickness, while fibrocartilage morphology and tendon area remained unchanged. Alendronate did not prevent structural bone loss, though it produced clear compositional changes such as increased carbonate substitution. Mineral to matrix ratio, crystallinity, and phosphate peak position did not differ significantly across any of the four treatment groups: under disuse- conditions, bisphosphonate treatment, or combined condition with bisphosphonate. Histomorphometry showed no differences in osteoblast or osteoclast number or surface, suggesting that bone loss occurs primarily through suppressed formation rather than elevated resorption. Because osteoclast number and surface were unchanged, elevated resorption is unlikely to account for observed bone loss. Osteoblast number and surface were similarly unaffected, indicating that any deficit in formation is more likely attributable to reduced activity per cell rather than to a reduction in osteoblast recruitment or abundance. Mechanical testing demonstrated that unloading reduced supraspinatus humerus complex strength and modulus. Although alendronate did not preserve trabecular structure, it produced a non-significant trend toward higher complex level strength, raising the possibility that material-level changes can influence failure independently of architecture. However, combined unloading and turnover suppression reduced post yield properties, highlighting tradeoffs for enthesis quality under low load conditions. These findings show that disuse induced bone loss at the supraspinatus enthesis is driven by formation and that bisphosphonates, while modifying mineral composition, do not prevent structural degeneration.

PubMedJournal of clinical medicine2026-09-15

Clinical and Radiological Outcomes of Oral Bisphosphonate Therapy Combined with Conservative Treatment in Subchondral Bone Marrow Edema of the Knee: A Retrospective Cohort Study.

Yurdakul Goker G, Korkmaz Murat M, Karadak Irfan I, Iyigun Abdullah A et al.

Background/Objectives: Subchondral bone marrow edema (BME) of the knee often causes persistent pain despite conservative treatment, and evidence supporting oral bisphosphonates is limited. We compared conservative treatment alone with conservative treatment plus oral alendronate. Methods: This retrospective cohort study included 82 patients with MRI-confirmed subchondral BME of the knee treated between January 2020 and January 2025. Patients received conservative treatment alone (n = 44) or conservative treatment plus alendronate 70 mg weekly for three months (n = 38). Visual Analog Scale (VAS) and Western Ontario and McMaster Universities Osteoarthritis Index (WOMAC) scores were assessed at baseline and three months. Radiological response was assessed on follow-up MRI as the percentage reduction in BME area. Results: Both groups improved significantly at three months. The alendronate group showed greater reductions in VAS (4.0 ± 1.4 vs. 3.2 ± 1.3 points, p = 0.009) and total WOMAC scores (25.5 ± 8.9 vs. 19.8 ± 8.6 points, p = 0.002). Median time to full weight-bearing was shorter with alendronate (7 vs. 9 weeks, p = 0.021), and complete BME regression was more frequent (42.1% vs. 18.2%, Fisher's exact p = 0.028). After adjustment for Kellgren-Lawrence grade, alendronate was associated with a favorable radiological response (OR = 3.66, 95% CI: 1.18-11.31, p = 0.024). No serious adverse events were recorded. Conclusions: Adding oral alendronate to conservative treatment was associated with greater clinical improvement, earlier full weight-bearing, and a more favorable radiological response at three months. Prospective randomized studies are needed to confirm these findings.

PubMedMacromolecular bioscience2026-09-15

Enzyme-Responsive Composite Hydrogel Coating Containing Nano-HA/Nano-ZnO with Pro-Osteogenic and Anti-Osteoclastic Properties.

Yu Yue Y, Liu Chengde C, Li Yizheng Y, Ma Zihan Z et al.

The treatment of osteoporotic bone defects remains challenging due to the pathological microenvironment with impaired regenerative capacity and imbalanced bone formation and resorption. In this study, a methacrylated gelatin (GelMA) and methacrylated chitosan (CSMA) composite hydrogel coating is developed on poly(phthalazinone ether nitrile ketone) (PPENK), showing pro-osteogenic and anti-osteoclastic effects in vitro. Nano-zinc oxide (nano-ZnO), nano-hydroxyapatite (nano-HA), and phosphate-ester-functionalized alendronate (ALNP) are incorporated to confer antibacterial activity, promote osteogenesis, and enable enzyme-responsive release of anti-osteoporosis alendronate (ALN). SEM/EDS results confirm the homogeneous dispersion of the nanoparticles within the porous hydrogel. The stability of the hydrogel coating is verified by smooth scratch edges and no visible detachment in the cross-hatch test. ALN release experiments confirm that the release amount of ALN is positively correlated with alkaline phosphatase (ALP) concentration, indicating significant enzyme-responsive characteristics. In vitro studies show that the hydrogel coating on PPENK exhibits antibacterial activity. Nano-HA and nano-ZnO enhance the expression of osteogenic-related genes and proteins in MC3T3-E1 pre-osteoblasts. The released ALN inhibits the receptor activator of nuclear factor-κB ligand (RANKL)-induced differentiation of RAW264.7 macrophages into osteoclasts. Overall, this surface modification strategy integrates enzyme-responsive ALN release with composite hydrogel coating on PPENK, realizing antibacterial, osteogenic, and anti-osteoclastic effects in vitro.

PubMedJournal of molecular histology2026-09-10

Naringin attenuates orchidectomy-induced bone loss in male rats: evidence from bone remodeling and telomere-related indices.

Zhong Yelin Y, Wu Guoying G, Xiong Shishuo S, Zhang Yukai Y et al.

Androgen deficiency contributes to secondary osteoporosis in men, yet the effects of naringin in androgen-deficient male bone loss remain unclear. We evaluated whether naringin attenuates orchidectomy (ORX)-induced bone loss in male Sprague-Dawley rats and whether this response is accompanied by changes in bone turnover, the OPG/RANKL axis, TERT expression, and relative telomere length. Forty rats were assigned to Sham, ORX, ORX + Naringin, and ORX + Alendronate (ALN) groups. Treatments were administered by oral gavage for 8 weeks. ORX impaired L4 trabecular microarchitecture, shifted serum bone turnover markers toward resorption, reduced the OPG/RANKL ratio, lowered TERT expression, and shortened relative telomere length. Naringin improved BV/TV, Tb.N, Tb.Th, and Tb.Sp; increased OCN and PINP; reduced CTX-I and TRACP-5b; and partially restored the OPG/RANKL balance compared with untreated ORX rats. Naringin was also associated with higher TERT expression and longer relative telomere length, whereas alendronate produced comparable structural and turnover responses but weaker telomere-related changes. These findings suggest that naringin attenuates ORX-induced bone loss in male rats, with coordinated changes in skeletal remodeling and telomere-related indices.

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